Notably, the authors did not find differences in tumor growth when they injected B16F10 or B16-BL6 cells (both of which express low MHC-I) in WT and TNFR1 KO mice, suggesting that expression of MHC-I at the surface of melanoma is important for growth inhibition induced by the lack of TNFR1 in the host [15]. Here, TNFRSF1A is linked to melanoma.