For instance, if the transcriptomics remodeling of some of the analyzed genes are effectively translated into the protein level, one can envisage both positive [anti-atherogenic properties (e.g., ApoA1 upregulation) and hepatic steatosis reversal (e.g., SCD-1 upregulation)] and negative [altered xenobiotic metabolism (e.g., acetaminophen, isoniazid, coumarin, ethanol), due to Cyp2E1 downregulation] outcomes [142,143,144]. This evidence concerns the gene APOA1 and fatty liver disease.