Among these, Bmp10, which has shown to induce cardiomyocyte proliferation, preserve cardiac function following myocardial infarction, and prevent cardiomyocyte death and development of cardiac fibrosis following insult,81, 82 was down‐regulated 30‐fold in tumour hosts, whereas ACVR2B/Fc increased Bmp10 five‐fold in tumour hosts. This evidence concerns the gene ACVR2B and myocardial infarction.