Perhaps more interesting, despite unchanged mitochondrial proteins, we did observe a benefit in assessment of oxidative metabolism in tumour mice treated with ACVR2B/Fc, whereby SDH enzyme activity and SDH staining of the tibialis anterior were improved, and PDH activity was unchanged in ACVR2B/Fc‐treated tumour mice compared with sham animals (Figure5). This evidence concerns the gene ACVR2B and neoplasm.