FMR1 and fragile X-associated tremor/ataxia syndrome: Knockdown of Schlank and Sk2 resulted in enhancement of premutation CGG repeat-mediated neurodegeneration in Drosophila, indicating that Schlank and Sk2 interact with the CGG repeat of FMR1 and act as genetic modifiers in the neurodegeneration of FXTAS (Kong et al., 2019).