Among these mRNAs, TGFBI was investigated as a highly induced transcript during EMT in the non-small-cell lung cancer cell line A549, acting as a competing endogenous RNA (ceRNA) (Qi et al., 2015) for miR-21 to modulate EMT, which indicated that the TGFBI 3′ UTR containing the miR-21 binding site could reduce miR-21 expression and mitigate its biological function (Liu et al., 2019). The gene discussed is TGFBI; the disease is non-small cell lung carcinoma.