Nevertheless, in a follow-up study, it has been shown that there is a certain redundancy in the CXCL10/CXCR3 axis since the reduction of T1D in CXCR3-deficient RIP-LCMV mice and in anti-CXCL10 antibody treated RIP-LCMV mice was not as pronounced as in earlier studies (Coppieters et al., 2013). The gene discussed is CXCL10; the disease is type 1 diabetes mellitus.