It has been suggested to prevent the viral entry, transport, and post-entry events in COVID-19, most likely via its effects on endosomal pH and the resulting under-glycosylation of angiotensin-converting enzyme 2 (ACE2) receptors that are required for viral entry, although the exact mechanisms remain incompletely understood (Roden et al., 2020; Singh et al., 2020). Here, ACE2 is linked to COVID-19.