IL1A and infection: Inhibits neuraminidase activity, reduces virus replication, and decreases the protein expression of TLR3 and NF-κB and mRNA expression of TRIF, the protein and gene expression of proinflammatory cytokines TNF-α, IL-1 and IL-6 in lung tissue, and increases the protein and gene expression of anti-inflammatory cytokine IL-10 and antiviral factor IFN-γ in lung tissue after infection.