The expression of antiapoptotic gene Bcl-2 in the prostates of mice in the BPH group was significantly upregulated, while the expression of apoptosis-related genes, such as Bax, p53, and caspase-3, were downregulated, resulting in cellular proliferation and BPH occurrence [16, 17]. The gene discussed is BCL2; the disease is benign prostatic hyperplasia.