In vivo experiments showed that GXV increases the ejection fraction and fractional shortening and reduces the left ventricular mass index [14, 15, 18] and reduces the levels of IL-6, TNF-α and other inflammatory factors [14, 15] in rats with acute myocardial infarction by inhibiting the NF-κB pathway [18, 19]. This evidence concerns the gene TNF and acute myocardial infarction.