Researchers indicated that autophagy is modulated dose-dependently by miRNA96 through regulation of mTOR and ATG7 required for the efficient formation of autophagosomes and suggested that the inhibition of mTOR by upregulation of miRNA-96 may promote autophagy in prostate cancer, which is involved in maintaining a dynamic balance of miRNA 96 in hypoxia [32]. The gene discussed is MTOR; the disease is prostate carcinoma.