FASLG and infection: Regarding Epstein–Barr virus (EBV)-infection, NKG2D receptor triggering and the cytotoxic pathways of TRAIL and Fas/Fas ligand (FasL) were outlined as the major mechanisms involved in clearing EBV-transformed autologous lymphoblastoid B cell lines by Vγ9Vδ2 T lymphocytes [79].