Furthermore, in an IAV-infected MDMs model, virus-activated Vγ9Vδ2 T cells driven from PBMs of healthy donors were shown to preferentially express the chemokine receptors CCR1, CCR5, and CXCR5, among which the CCR5 is associated with the regulation of Vγ9Vδ2 T cell trafficking to virus-infected cells by binding to its ligands (CCL3, CCL4, and CCL5) produced in response to influenza infection. This evidence concerns the gene CCR5 and influenza.