ARMS has a poor prognosis [2] because ARMS cells harbor chromosomal translocation t(2;13)(q35;q14) or t(1;13)(q35;q14), which results in the formation of the characteristic chimeric gene through the fusion of PAX3 or PAX7 with FOXO1 that is involved in the pathogenesis of ARMS [3,4,5,6]. This evidence concerns the gene FOXO1 and alveolar rhabdomyosarcoma.