Interestingly, induction of P53 resulted in increased expression of colony-stimulating factor 1 (CSF1), CCL2, CXCL1, and IL-15 as well as of the adhesion molecules intercellular adhesion molecule 1 (ICAM1) and vascular cell adhesion molecule 1 (VCAM1), which further recruit natural killer (NK) cells to trigger tumor regression, arguing that oncogenic pathways might also influence immune cell types. The gene discussed is CSF1; the disease is neoplasm.