HAVCR1 and primary hyperoxaluria type 1: Renal injury (β2-microglobulin, laminin α5, and NGAL) marker-positive urinary EVs were statistically (Q < 0.20) different between PH1 patients without and with NC or KS, whereas the number of clusterin and kidney injury molecule-1 (KIM-1)-carrying urinary EV did not differ between groups (Table 3).