CD36 and metabolic dysfunction-associated steatohepatitis: In addition, recent studies suggest that inflammatory stress increases hepatic CD36 protein level via activation of the mTOR pathway to enhance its translational efficiency.[56] Moreover, CD36 was highly palmitoylated in mice with NASH and palmitoylation was shown to help it translocate to the plasma membrane of hepatocytes, thereby facilitating fatty acid uptake.[40] Therefore, together with these studies, our data suggest that the expression, translocation, and activity of CD36 are rather complicated in the progression of chronic liver disease.