SOD1 and neoplasm: The tumor acidic condition decomposed copper peroxide to release Cu ions, which acted as the catalysts for converting self‐supplying H2O2 into highly toxic hydroxyl radicals and consequently inhibiting tumor growth.[108] Similarly, the co‐attachment of superoxide dismutase (SOD) and Cu component into calcium carbonate (CaCO3)‐mineralized nanoparticles also achieved the production of hydrogen peroxide by SOD and efficient Fenton‐like reaction as catalyzed by the doped Cu component, resulting in the hydroxyl radicals production and specific toxicity to cancer cells.[109]