However, it is worth noting thatthe authors somewhat self-confidently declared that there is a role for Cx32 inregulating the sensitivity of tumor cells to chemotherapy and the possibilityof using it as a target for therapy based only on the potential relationshipbetween the phenotype and sensitivity, while there were no relevant experimentsconfirming the sensitization of doxorubicin to cells with Cx32 overexpression[153]. The gene discussed is GJB1; the disease is neoplasm.