Different mechanisms underlying the antitumor action of MA in gastric cancer cells have been shown, including cell cycle arrest at G1 and G2/M phase, apoptosis induction either via increasing proapoptotic molecules of Bcl-2 family (the intrinsic pathway) or upregulation of Fas (CD95/APO-1)/Fas ligand (FasL) expression (the extrinsic pathway) and autophagy associated with upregulation of Beclin-1 expression. This evidence concerns the gene FASLG and gastric cancer.