HDAC9 and retinitis pigmentosa 1: Consequently, in the view of the use of HDAC inhibition for the treatment of various diseases, and our previous data linking HDAC overactivation to rod loss in different mouse models of RP, this study highlights HDACs as a common denominator of both mutation-induced rod cell death and secondary cone degeneration and provides a unique therapeutic option for the treatment of RP regardless of the stage of degeneration.