The use of a custom panel consisting of a smaller subset of genes associated with necroptosis and pyroptosis as well as pyroptosis-related inflammasomes in this report also served to confirm our previous determination that these programmed cell death pathways appear to be involved in the progression of MCMV retinitis during MAIDS [62] as evidenced by this earlier report of significant intraocular stimulation of necroptosis-associated receptor interacting protein kinase 3 (RIPK3), pyroptosis-asssociated caspase-1, interleukin-1β, interleukin-18, and the AIM2 inflammasome. This evidence concerns the gene CASP1 and retinitis.