In our prior work using the same model, we explored the components of the canonical transcription factor NF-κB (dorsal, Dif, Relish) and a wider panel of receptors, signaling molecules, and antimicrobial peptides (PGRP-SD, Toll, Metchnikowin, Cecropin A, JNK, Drosomycin, Defensin, InR, IRS, PTEN, Akt1, Foxo, mTORC1, and ratio of p-Akt/Akt) as surrogates for sustained inflammation among flies surviving sepsis. The gene discussed is TLR4; the disease is Sepsis.