Similarly, the two IFN-associated modules, HB12 and HB23, also previously reported as over-abundant in the blood from patients with active TB disease35 and from mouse models of increased TB susceptibility16, were over-abundant in the blood from infected Ctrl Ab treated mice and further increased in the blood from αGM-CSF treated mice (Fig. 2a, b). The gene discussed is IFNA1; the disease is tuberculosis.