The authors confirmed these results in a later study, investigating the co-deposition in myocardial infarction of MAC with clusterin, a plasma glycoprotein involved in the regulation of MAC activation, reporting also the activity of this protein, as well as other regulators, in the clearance of the damaged tissue rather than in the protection of the myocardial cells from full assembly of the MAC [37]. The gene discussed is CLU; the disease is myocardial infarction.