D1R activation in triple-negative breast cancer cell line (4T1.2) leads to the induction of apoptosis (increased level of cleaved caspase 3), autophagy (elevation of LC3-phosphatidylethanolamine conjugate–LC3A/B II), and the phosphorylation of eukaryotic translation initiation factor 2-alpha (eIF2a) [55,72,73]. The gene discussed is MAP1LC3A; the disease is triple-negative breast carcinoma.