In the abdominal aorta from C57BL/6 mice with CaCl2/PBS-induced AAA, the expression levels of UBF, TBP and RPA43 (the TIF-IA interacting subunit of Pol I) [25] were not altered at day 7, while the level of TIF-IA was reduced (Figure 1A). The gene discussed is RRN3; the disease is triple-A syndrome.