ATP, at concentrations (200 μm) chosen to be within the extracellular physiological concentrations previously found (> 100 μm) in the lung cancer cell lines used in this study [1, 2, 3], was found to weaken interactions between AChE and Aβ (Fig. 3) but strengthens those between Aβ and HN (Fig. 4). This evidence concerns the gene ACHE and lung carcinoma.