In agreement with Edu results, silencing eEF-2K caused an accumulation of cells in the G0/G1 population and a reduction in S and G2/M population in everolimus-treated breast cancer cells (Fig. 3D), indicating that suppression of eEF-2K further caused G0/G1 cell cycle arrest induced by mTOR inhibitor. The gene discussed is EEF2K; the disease is breast carcinoma.