EZH1 has been previously reported to be a target gene of miR-17-5p, and the down-regulation of miR-17-5p could enhance the resistance to erlotinib in non-small cell lung cancer by upregulating EZH1 (Zhang et al. 2017), which was in consistent with our experimental results whereby miR-17 in the miR-17-92 cluster negatively regulated EZH1 expression, which reflects the biological characteristics of BPD. Here, EZH1 is linked to bronchopulmonary dysplasia.