HDAC3 and bronchopulmonary dysplasia: In terms of the degree of abnormal alveolarization in mice, the MLI of hyperoxia + Hdac3−/−-treated mice was reduced, and that of the hyperoxia + Hdac3−/− + miR-17-antagomir mice was similar to that of hyperoxia-induced BPD mice (Fig. 6c).