STING1 and triple-A syndrome: Lou et al. found, using the Angiotensin II model of AAA, that mice deficient in STING (Stinggt/gt) were protected from aortic aneurysm, dissection, and rupture, and that levels of phosphorylated RIPK3 and MLKL in treated aortic tissues were significantly decreased in STING deficient mice.