In a previous study, we retrospectively collected data on 268 patients with thymomatous MG, and selected patients with symptoms of spontaneous muscle overactivity for autoantibody testing for neuronal cell‐surface proteins and cell‐based assays for contactin‐associated protein 2 (CASPR2), leucine‐rich glioma inactivated 1 (LGI1), glycine receptor and Netrin‐1 receptor antibodies. This evidence concerns the gene CNTNAP2 and myasthenia gravis.