IL1B and acute respiratory distress syndrome: A cohort study of 192 COVID-19 patients showed that increased IL-6 and IL-10 and decreased CD4+ T cells were independent risk factors for severe liver damage.73 High levels of IL-1β, IL-2, IL-6, IL-8, IL-10, IL-17, interferon, IP10, and monocyte chemoattractant protein 1 in patients infected with SARS-CoV-2 have also been observed.74,76 Much release of inflammatory cytokines by immune cells triggers acute respiratory distress syndrome (ARDS) and SIRS.