In addition to our findings regarding the CXCL10-CXCR3 axis, we observed that the released amount of mediators involved in chemotaxis and/or activation of PMNs (i.e., IL-8, IL-1β, IL-6, and TNF-α) was similar when ARDS was caused by SARS-CoV-2. The gene discussed is TNF; the disease is acute respiratory distress syndrome.