To explore if any NRAS-related transcriptomic patterns observed in hPheo1 cells are present in PPGL tumour samples, we analysed cDNA hybridization microarray data on 26 samples with known driver mutations from a Scandinavian patient cohort [36] (including 5 EPAS1-mutated PPGL belonging to a pseudohypoxic subtype; and 11 NF1-mutated, 5 HRAS-mutated, 3 RET-mutated, and 2 FGFR-mutated PPGLs belonging to RTK-driven subtypes) and RNA-sequencing data on 186 samples from The Cancer Genome Atlas (TCGA) project [14]. The gene discussed is HRAS; the disease is neoplasm.