Further, they present data showing that radiation therapy can induce fibroblast growth factor receptor (FGFR) family-mediated PTENTyr-240 phosphorylation and co-recruitment of Ki-67 and RAD51 to chromatin in PTEN-proficient GBM, thus enhancing DSBR activity and radioresistance in GBM patient-derived stem-like cells (GSCs). This evidence concerns the gene MKI67 and glioblastoma.