CCL24 and infection: Starting at 5 days post-transmission, and especially by 12 days, the infections transitioned into mainly inflammatory cells, although there was considerable variability in the myeloid subsets infected, emphasizing the heterogeneity of natural transmission sites even at 1–2 weeks p.i. At 5 days, a few of the ears showed a relatively high proportionate number of infected eosinophils, extending their influx to natural transmission sites, which was shown to be mediated by CCL24 produced by dermal TRM following infection by needle [28].