LDLR and familial hyperaldosteronism: Even though FH is thought to be caused by deleterious mutations in genes associated with LDL metabolism such as LDL receptor (LDLR), apolipoprotein B (APOB), and proprotein convertase subtilisin/kexin type 9 (PCSK9), and accumulation of LDL-raising single nucleotide polymorphisms [2], genetic testing is rarely used in clinical settings [3].