Both MYC and SNHG12 acted in a similar fashion in NKTCL by promoting proliferation, enhancing the level of P-glycoproteins (P-gp) that are linked to multi drug resistance (MDR) proteins and inhibiting the sensitivity to cisplatin (CCDP) (94). The gene discussed is SNHG12; the disease is extranodal nasal NK/T cell lymphoma.