ZFP69 and Hyperglycemia: DBA mice carry an intact copy of Zfp69, however, characterization of recombinant congenic mice excluded the possibility that Zfp69 is responsible for β-cell failure observed in Nidd/DBA mice, as Zfp69 is located outside of the smallest critical Nidd/DBA fragment (108.1–111.4 Mbp) inducing hyperglycemia.