In growing cancers, endothelial cells are vigorously active as a result of the release of proteins including epidermal growth factor, estrogen, basic and acidic fibroblast growth factors, interleukin-8, tumor necrosis factor−α, and vascular endothelial growth factor (VEGF), which can activate endothelial cell growth and motility when the production of antiangiogenic factors is reduced [6,7]. This evidence concerns the gene VEGFA and cancer.