TNF and COVID-19: Following capture of sterile antigens (necrotic cardiomyocytes and damaged fragments from ischemic myocardium) in STEMI and viruses after COVID-19 principally by monocytes and dendritic cells, there is massive secretion of proinflammatory molecules (e.g., interleukin-1, interleukin-6, tumor necrosis factor-α, among others).