XIST and non-small cell lung carcinoma: For example, via suppressing autophagy, the chemosensitivity of NSCLC cells is enhanced by silencing lncRNA-XIST, and through regulating the WNT pathway in NSCLC and repressing E-cadherin, epithelial-mesenchymal transition (EMT) is enhanced by the overexpression of FEZF1-AS1 [14].