Nuclear paraspeckle assembly transcript 1 (NEAT1) is proven to use NACHT, LRR, and PYD domain-containing protein 3 (NLRP3) inflammasomes as molecular decoys for miR-3076-3p, so knockdown NEAT1 could facilitate the tolerogenic phenotype in DC, which prevents progression of experimental autoimmune myocarditis and induces immune tolerance in a heart transplantation model (70). The gene discussed is NEAT1; the disease is autoimmune myocarditis.