These results indicate that MCT2 is associated with PCa proliferation and, more specifically, that the peroxisomal localization of MCT2 seems to be required for PCa proliferation as, upon inhibition peroxisome membrane protein by PEX19 knockdown, MCT2 overexpression was not able to elevate the cells’ proliferative capacity. This evidence concerns the gene SLC16A7 and posterior cortical atrophy.