STK35 and neoplasm: Previous studies have reported that STK35 and its homolog CLP36 Interacting Kinase 1 Like (Goyal et al., 2009) are capable of regulating a variety of cellular activities in human cancers, in which they have been shown to promote cellular proliferation, migration, and invasion (Yasuda et al., 2012), stimulate the metabolic processes of cancer cells (Zhang et al., 2017), and suppress tumor cell apoptosis (Wu et al., 2018).