Subsequent evidence, such as the observation of the surface localization of HSP90AA1 and IAV HA1 (Figure 1C), and the results that blocking cell surface HSP90AA1 with anti-HSP90AA1 antibodies and treating H1N1 viruses with purified HSP90AA1 protein both substantially inhibited attachment of virus on the cell membrane and reduced virus infection greatly (Figure 2), confirming that HSP90AA1 participated in the process of influenza virus attachment. The gene discussed is HSP90AA1; the disease is viral infectious disease.