The expression of several CEBPα and PPARγ target genes, such as Atgl, Hsl, and perilipin (lipolysis-related genes); Lpl, lipin, Dgat1, and Dgat2 (fatty acid uptake and lipogenesis-related genes); leptin and Nos2 (obesity inflammation-related genes); and Cpt1, Nrf1, Tfam, and Prdm16 (fatty acid oxidation and biogenesis-related genes) were also partially reversed in the ING fat depots of animals treated with palmitoleic acid. This evidence concerns the gene LEP and obesity due to melanocortin 4 receptor deficiency.