MM patients were categorised according to their molecular subgroups, defined by Zhan and coworkers [21] as proliferating (PR), low bone disease (LB), with activated FGFR3/MMSET genes (MS), hyperdiploid (HY), expressing cyclin D1 (CD1/2), with activated MAF gene (MF). This evidence concerns the gene CCND1 and Miyoshi myopathy.