ATF4 and myocardial infarction: Fbxl10 expression reduced myocardial infarction, remodeling, and inflammatory response in myocardial ischemia-reperfusion rats by suppressing the expression of endoplasmic reticulum stress proteins including DNA damage-inducible transcript 3 (Ddit3, also known as Chop), heat shock protein family A member 5 (Hspa5, also known as Grp78), activating transcription factor 4 (Atf4, also known as Creb2), and phospho-Erk (mitogen-activated protein kinase) [122].