In accordance with the aforementioned study, CD11c+CD11b+ cells isolated from mice following Ova-specific Th2 cell transfer and allergen challenge, produced more CCL17 and CCL22 compared to CD11c+/CD11b− cells from the same mice underlying the importance of mo-DCs in recruiting other effector subsets during the effector phase of experimental asthma [61]. The gene discussed is ITGAM; the disease is asthma.